"Mice, Inbred C57BL" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
One of the first INBRED MOUSE STRAINS to be sequenced. This strain is commonly used as genetic background for transgenic mouse models. Refractory to many tumors, this strain is also preferred model for studying role of genetic variations in development of diseases.
Descriptor ID |
D008810
|
MeSH Number(s) |
B01.050.050.199.520.520.420 B01.050.150.900.649.313.992.635.505.500.400.420
|
Concept/Terms |
Mice, Inbred C57BL- Mice, Inbred C57BL
- C57BL Mice, Inbred
- Inbred C57BL Mice
- Mouse, Inbred C57BL
- C57BL Mouse, Inbred
- Inbred C57BL Mouse
- Mice, C57BL
- C57BL Mice
- Mouse, C57BL
- C57BL Mouse
|
Below are MeSH descriptors whose meaning is more general than "Mice, Inbred C57BL".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred C57BL".
This graph shows the total number of publications written about "Mice, Inbred C57BL" by people in this website by year, and whether "Mice, Inbred C57BL" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1998 | 0 | 2 | 2 |
2002 | 0 | 1 | 1 |
2004 | 0 | 1 | 1 |
2005 | 0 | 1 | 1 |
2006 | 0 | 3 | 3 |
2008 | 0 | 6 | 6 |
2009 | 0 | 10 | 10 |
2010 | 0 | 3 | 3 |
2011 | 0 | 10 | 10 |
2012 | 0 | 7 | 7 |
2013 | 0 | 18 | 18 |
2014 | 0 | 12 | 12 |
2015 | 0 | 19 | 19 |
2016 | 0 | 14 | 14 |
2017 | 0 | 14 | 14 |
2018 | 0 | 18 | 18 |
2019 | 0 | 14 | 14 |
2020 | 0 | 19 | 19 |
2021 | 0 | 11 | 11 |
2022 | 0 | 7 | 7 |
2023 | 0 | 3 | 3 |
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Below are the most recent publications written about "Mice, Inbred C57BL" by people in Profiles.
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Unleashing T cell anti-tumor immunity: new potential for 5-Nonloxytryptamine as an agent mediating MHC-I upregulation in tumors. Mol Cancer. 2023 08 15; 22(1):136.
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IL-6 in the infarcted heart is preferentially formed by fibroblasts and modulated by purinergic signaling. J Clin Invest. 2023 06 01; 133(11).
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Activation of ?-adrenergic receptor signaling prevents glucocorticoid-induced obesity and adipose tissue dysfunction in male mice. Am J Physiol Endocrinol Metab. 2023 06 01; 324(6):E514-E530.
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IGF1R expression by adult oligodendrocytes is not required in the steady-state but supports neuroinflammation. Glia. 2023 03; 71(3):616-632.
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Cardiomyocyte p38 MAPKa suppresses a heart-adipose tissue-neutrophil crosstalk in heart failure development. Basic Res Cardiol. 2022 10 07; 117(1):48.
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Perinatal Obesity Induces Hepatic Growth Restriction with Increased DNA Damage Response, Senescence, and Dysregulated Igf-1-Akt-Foxo1 Signaling in Male Offspring of Obese Mice. Int J Mol Sci. 2022 May 17; 23(10).
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Voluntary Wheel Running in Old C57BL/6 Mice Reduces Age-Related Inflammation in the Colon but Not in the Brain. Cells. 2022 02 06; 11(3).
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Exosomal miRNAs from Prostate Cancer Impair Osteoblast Function in Mice. Int J Mol Sci. 2022 Jan 24; 23(3).
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Human Leucocyte Antigen G and Murine Qa-2 Are Critical for Myeloid Derived Suppressor Cell Expansion and Activation and for Successful Pregnancy Outcome. Front Immunol. 2021; 12:787468.
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Gene-selective transcription promotes the inhibition of tissue reparative macrophages by TNF. Life Sci Alliance. 2022 04; 5(4).